KPV
Modulates the inflammatory environment
- What it is
- The C-terminal tripeptide (Lys-Pro-Val) of α-MSH.
- Studied for
- Studied for inhibition of NF-κB nuclear translocation, which is the transcription factor that drives pro-inflammatory cytokine expression. Research models focus on gastrointestinal and general inflammatory signalling.
- Why it's in this stack
- A persistent inflammatory signal is the usual confounder in repair models. KPV is the arm of the stack studied at that upstream step.



